Bell palsy
Idiopathic (likely viral) lower-motor-neuron CN VII palsy
Overview
An acute, idiopathic (likely post-viral, HSV-linked) LOWER-motor-neuron facial nerve palsy. The key discriminator is that it involves the WHOLE side of the face including the FOREHEAD (forehead-sparing implies an UMN/central cause such as stroke). Early prednisolone improves recovery.
Recognise
- Acute (over hours–days) unilateral facial weakness involving the FOREHEAD (cannot raise the eyebrow / close the eye)
- May have post-auricular pain, altered taste, hyperacusis, dry eye
- Forehead INVOLVED = lower motor neuron (Bell); forehead SPARED = upper motor neuron (stroke)
Red flags
- Forehead sparing (central cause — stroke), bilateral palsy, other cranial nerves, vesicles (Ramsay Hunt), no recovery → reconsider
Differentials & how to tell them apart
Investigations
Clinical diagnosis. Examine for forehead sparing (UMN) and a vesicular rash (Ramsay Hunt = herpes zoster); consider Lyme/parotid causes if atypical.
Management
Prednisolone within 72h + meticulous eye protection
- 1Confirm it is lower-motor-neuron (forehead involved). Start prednisolone within 72 hours; protect the eye (lubricants, taping at night).Gate: Forehead SPARING means an upper-motor-neuron lesion (e.g. stroke) — that is NOT Bell palsy and needs urgent stroke assessment
- 2Add antivirals if Ramsay Hunt/zoster; ophthalmology if incomplete eye closure; most recover over weeks.
Key points
Forehead involved = Bell (LMN); forehead spared = stroke (UMN) — the single most important discriminator. Eye protection prevents exposure keratopathy. Steroids within 72h.
Monitor & prognosis
Recovery, corneal protection/eye health.
Most recover fully within weeks; steroids improve the odds.
Source: NICE CKS Bell palsy