The drug atlas
Cardiovascular/Dihydropyridine CCB

Dihydropyridine CCBs

also: DHP · amlodipine class

Overview

Vascular-selective calcium-channel blockers. First-line for hypertension (esp. age ≥55 or Black African/Caribbean) and for stable angina.

Mechanism

Block L-type voltage-gated Ca²⁺ channels on vascular smooth muscle → ↓Ca²⁺ influx → arteriolar vasodilation → ↓systemic vascular resistance/afterload. Vascular-selective: negligible negative inotropy/chronotropy at therapeutic doses (unlike non-DHPs).

Indications

  • Hypertension
  • Stable angina
  • Raynaud phenomenon
  • Nifedipine: tocolysis (off-label)

The agents

Amlodipinelong-acting

t½ 35–50 h, once daily

Most-used; smooth 24 h cover; HTN + angina.

Nifedipineshort / MR

MR preparations only

Tocolysis, Raynaud. Avoid immediate-release for angina/HTN — reflex tachycardia.

Felodipinelong-acting

OD

Strong CYP3A4 substrate — marked grapefruit interaction.

Lercanidipinelong-acting

OD

Less ankle oedema than amlodipine (newer DHP).

Adverse effects

Ankle / peripheral oedemaclassic

Precapillary arteriolar dilation — NOT fluid overload; does not respond to diuretics.

Flushingcommon
Headachecommon
Reflex tachycardia / palpitationscommon

Worst with short-acting nifedipine.

Gingival hyperplasiaclassic

Cautions & contraindications

Severe aortic stenosisall

Vasodilation drops coronary perfusion → syncope/collapse.

Unstable angina, recent MIall

Reflex tachycardia ↑myocardial O₂ demand (short-acting).

Cardiogenic shockall

Interactions

  • CYP3A4 substrate — grapefruit juice raises levels
  • Amlodipine + simvastatin: cap simvastatin at 20 mg/day (myopathy risk)

Monitoring & kinetics

Blood pressure,Peripheral oedema (clinical)

Oral; hepatic CYP3A4 metabolism. Safe in renal impairment (no dose adjustment).

Source: BNF — Calcium-channel blockers · NICE NG136 — Hypertension